| [1]Lockshin RA, Williams CM. Programmed cell death I. Cytology of degeneration in intersegmental muscles of silkmoths. J Insect Physiol, 1964, 10: 123-133.
[2]Kerr JF, Wyllie AH, Currie AR. Apoptosis: a basic biological phenomenon with wide-ranging implications in tissue kinetics. Br J Cancer, 1972, 26(4): 239-257.
[3]Schweichel JU, Merker HJ. The morphology of various types of cell death in prenatal tissues. Teratology, 1973, 7(3): 253-266.
[4]Tsuda H, Ning Z, Yamaguchi Y, Suzuki N. Programmed cell death and its possible relationship with periodontal disease. J Oral Sci, 2012, 54(2): 137-149.
[5]Vanden Berghe T, Vanlangenakker N, Parthoens E, Deckers W, Devos M, Festjens N, Guerin CJ, Brunk UT, Declercq W, Vandenabeele P. Necroptosis, necrosis and secondary necrosis converge on similar cellular disintegration features. Cell Death Differ, 2010, 17(6): 922-930.
[6]Galluzzi L, Vitale I, Abrams JM, Alnemri ES, Baehrecke EH, Blagosklonny MV, Dawson TM, Dawson VL, El-Deiry WS, Fulda S, Gottlieb E, Green DR, Hengartner MO, Kepp O, Knight RA, Kumar S, Lipton SA, Lu X, Madeo F, Malorni W, Mehlen P, Nuñez G, Peter ME, Piacentini M, Rubinsztein DC, Shi Y, Simon HU, Vandenabeele P, White E, Yuan J, Zhivotovsky B, Melino G, Kroemer G. Molecular definitions of cell death subroutines: recommendations of the Nomenclature Committee on Cell Death 2012. Cell Death Differ, 2012, 19(1): 107-120.
[7]王连邦, 张丽英, 单春华. 一个新的细胞死亡形式: Parthanatos. 遗传, 2010, 32(9): 881-885.
[8]Kaiser WJ, Upton JW, Long AB, Livingston-Rosanoff D, Daley-Bauer LP, Hakem R, Caspary T, Mocarski ES. RIP3 mediates the embryonic lethality of caspase-8-deficient mice. Nature, 2011, 471(7338): 368-372.
[9]Mocarski ES, Upton JW, Kaiser WJ. Viral infection and the evolution of caspase 8-regulated apoptotic and necrotic death pathways. Nat Rev Immun, 2011, 12(2): 79-88.
[10]Vanlangenakker N, Vanden Berghe T, Bogaert P, Laukens B, Zobel K, Deshayes K, Vucic D, Fulda S, Vandenabeele P, Bertrand MJ. cIAP1 and TAK1 protect cells from TNF-induced necrosis by preventing RIP1/RIP3-dependent reactive oxygen species production. Cell Death Differ, 2011, 18(4): 656-665.
[11]Duprez L, Bertrand MJ, Vanden Berghe T, Dondelinger Y, Festjens N, Vandenabeele P. Intermediate domain of receptor-interacting protein kinase 1 (RIPK1) determines switch between necroptosis and RIPK1 kinase-dependent apoptosis. J Biol Chem, 2012, 287(18): 14863-14872.
[12]McQuade T, Cho Y, Chan FK. Positive and negative phosphorylation regulates RIP1-and RIP3-induced programmed necrosis. Biochem J, 2013, 456(3): 409-415.
[13]Dillon CP, Oberst A, Weinlich R, Janke LJ, Kang TB, Ben-Moshe T, Mak TW, Wallach D, Green DR. Survival function of the FADD-CASPASE-8-cFLIP(L) complex. Cell Rep, 2012, 1(5): 401-407.
[14]Vandenabeele P, Galluzzi L, Vanden Berghe T, Kroemer G. Molecular mechanisms of necroptosis: an ordered cellular explosion. Nat Rev Mol Cell Biol, 2010, 11(10): 700-714.
[15]Ikner A, Ashkenazi A. TWEAK induces apoptosis through a death-signaling complex comprising receptor- interacting protein 1 (RIP1), Fas-associated death domain (FADD) and caspase-8. J Biol Chem, 2011, 286(24): 21546-21554.
[16]Kaiser WJ, Sridharan H, Huang C, Mandal P, Upton JW, Gough PJ, Sehon CA, Marquis RW, Bertin J, Mocarski ES. Toll-like receptor 3-mediated necrosis via TRIF, RIP3, and MLKL. J Biol Chem, 2013, 288(43): 31268-31279.
[17]He S, Liang Y, Shao F, Wang X. Toll-like receptors activate programmed necrosis in macrophages through a receptor-interacting kinase-3-mediated pathway. Proc Natl Acad Sci USA, 2011, 108(50): 20054-20059.
[18]Mantel F, Frey B, Haslinger |
