[1] Liu TX, Becker MW, Jelinek J, Wu WS, Deng M, Mik-halkevich N, Hsu K, Bloomfield CD, Stone RM, DeAn-gelo DJ, Galinsky IA, Issa JP, Clarke MF, Look AT. Chromosome 5q deletion and epigenetic suppression of the gene encoding alpha-catenin (CTNNA1) in myeloid cell transformation. Nature Medicine, 2007, 13(1): 78-83.[2] Fu CT, Zhu KY, Mi JQ, Liu YF, Murray ST, Fu YF, Ren CG, Dong ZW, Liu YJ, Dong M, Jin Y, Chen Y, Deng M, Zhang W, Chen B, Breslin P, Chen SJ, Chen Z, Becker MW, Zhu J, Zhang JW, Liu TX. An evolutionarily con-served PTEN-C/EBP{alpha}-CTNNA1 axis controls myeloid development and transformation. Blood, 2010, 115(23): 4715-4724.[3] Zhou T, Wang L, Zhu KY, Dong M, Xu PF, Chen Y, Chen SJ, Chen Z, Deng M, Liu TX. Dominant-negative C/ebp {alpha} and polycomb group protein Bmi1 extends short-lived hematopoietic stem/progenitor cells lifespan and in-duces lethal dyserythropoiesis. Blood, 2011, 118(14): 3842-3852.[4] Fu YF, Du TT, Dong M, Zhu KY, Jing CB, Zhang Y, Wang L, Fan HB, Chen Y, Jin Y, Yue GP, Chen SJ, Chen Z, Huang QH, Jing Q, Deng M, Liu TX. Mir-144 selectively regulates embryonic {alpha}-hemoglobin synthesis during primitive erythropoiesis. Blood, 2009, 113(6): 1340-1349. |