[1] Harraz MM, Dawson TM, Dawson VL. Advances in neu-ronal cell death 2007. Stroke, 2008, 39(2): 286-288.
[2] Yu SW, Wang H, Poitras MF, Coombs C, Bowers WJ, Federoff HJ, Poirier GG, Dawson TM, Dawson VL. Me-diation of poly(ADP-ribose) polymerase-1-dependent cell death by apoptosis-inducing factor. Science, 2002, 297(5579): 259-263.
[3] Andrabi SA, Kim NS, Yu SW, Wang H, Koh DW, Sasaki M, Klaus JA, Otsuka T, Zhang Z, Koehler RC, Hurn PD, Poirier GG, Dawson VL, Dawson TM. Poly(ADP-ribose) (PAR) polymer is a death signal. Proc Natl Acad Sci USA, 2006, 103(48): 18308–18313.
[4] Yu SW, Andrabi SA, Wang H, Kim NS, Poirier GG, Dawson TM, Dawson VL. Apoptosis-inducing factor medi-ates poly(ADP-ribose) (PAR) polymer-induced cell death. Proc Natl Acad Sci USA, 2006, 103(48): 18314–18319.
[5] David KK, Andrabi SA, Dawson TM, Dawson VL. Par-thanatos, a messenger of death. Front Biosci, 2009, 14: 1116–1128.
[6] Edinger AL, Thompson CB. Death by design: apoptosis, necrosis and autophagy. Curr Opin Cell Biol, 2004, 16(6): 663–639.
[7] Kroemer G, El-Deiry WS, Golstein P, Peter ME, Vaux D, Vandenabeele P, Zhivotovsky B, Blagosklonny MV, Malorni W, Knight RA, Piacentini M, Nagata S, Melino G. Classification of cell death: recommendations of the No-menclature Committee on Cell Death. Cell Death Differ, 2009, 16(1): 3–11.
[8] Galluzzi L, Kroemer G. Necroptosis: A specialized path-way of programmed necrosis. Cell, 2008, 135(7): 1161–1163.
[9] Rosenbaum DM, Degterev A, David J, Rosenbaum PS, Roth S, Grotta JC, Cuny GD, Yuan J, Savitz SI. Necrop-tosis, a novel form of caspase-independent cell death, contributes to neuronal damage in a retinal ische-mia-reperfusion injury model. J Neurosci Res, 2010, 88(7): 1569-1576.
[10] Miao B, Degterev A. Methods to analyze cellular necrop-tosis. Methods Mol Biol, 2009, 559: 79–93.
[11] Andrabi SA, Dawson TM, Dawson VL. Mitochondrial and nuclear cross talk in cell death: parthanatos. Ann N Y Acad Sci, 2008, 1147: 233–241.
[12] Dawson VL, Dawson TM. Deadly conversations: nu-clear-mitochondrial cross-talk. J Bioenerg Biomembr, 2004, 36(4): 287–294.
[13] Wang Y, Dawson VL, Dawson TM. Poly(ADP-ribose) signals to mitochondrial AIF: A key event in parthanatos. Exp Neurol, 2009, 218(2): 193–202.
[14] Alano CC, Tran A, Tao R, Ying W, Karliner JS, Swanson RA. Differences among cell types in NAD(+) compartmentaliza-tion: A comparison of neurons, astrocytes, and cardiac myo-cytes. J Neurosci Res, 2007, 85(15): 3378–3385.
[15] Koh DW, Lawler AM, Poitras MF, Sasaki M, Wattler S, Nehls MC, Stöger T, Poirier GG, Dawson VL, Dawson TM. Failure to degrade poly(ADP-ribose) causes increased sensitivity to cytotoxicity and early embryonic lethality. Proc Natl Acad Sci USA, 2004, 101(51): 17699–17704.
[16] Polster BM, Basañez G, Etxebarria A, Hardwick JM, Nicholls DG. Calpain I induces cleavage and release of apoptosis-inducing factor from isolated mitochondria. J Biol Chem, 2005, 280(8): 6447–6454.
[17] Cao G, Xing J, Xiao X, Liou AK, Gao Y, Yin XM, Clark RS, Graham SH, Chen J. Critical role of calpain I in mi-tochondrial release of apoptosis-inducing factor in ischemic neuronal injury. J Neurosci, 2007, 27(35): 9278–9293.
[18] Moubarak RS, Yuste VJ, Artus C, Bouharrour A, Greer PA, Menissier-de Murcia J, Susin SA. Sequential activation of poly(ADP-ribose) polymerase 1, calpains, and Bax is es-sential in apoptosis-inducing factor-mediated programmed necrosis. Mol Cell Biol, 2007, 27(13): 4844–4862.
[19] Vosler PS, Sun D, Wang S, Gao Y, Kintner DB, Signore AP, Cao G, Chen J. Calcium dysregulation induces apop-tosis-inducing factor release: cross-talk between PARP-1- and calpain-signaling pathways. Exp Neurol, 2009, 218(2): 213–220.
[20] Wang |