[1] Cantoni C, Falco M, Pessino A, Moretta A, Moretta L, Biassoni R. P49, a putative HLA-G1 specific inhibitory NK receptor belonging to the immunoglobulin Superfamily. J Reprod Immunol, 1999, 43(2): 157-165.[2] Hviid TVF, Hylenius S, Rørbye C, Nielsen LG. HLA-G allelic variants are associated with differences in the HLA-G mRNA isoform profile and HLA-G mRNA levels. Immunogenetics, 2003, 55(2): 63-79.[3] Larsen MH, Hviid TVF. Human leukocyte antigen-G polymorphism in relation to expression, function, and disease. Hum Immunol, 2009, 70(12): 1026-1034.[4] Carosella ED, Moreau P, Lemaoult J, Rouas-Freiss N. HLA-G: from biology to clinical benefits. Trends Immunol, 2008, 29(3): 125-132.[5] Rousseau P, Le Discorde M, Mouillot G, Marcou C, Carosella ED, Moreau P. The 14 bp deletion-insertion polymorphism in the 3' UT region of the HLA-G gene influences HLA-G mRNA stability. Hum Immunol, 2003, 64(11): 1005-1010.[6] Aruna M, Sirisha PVS, Bhaskar S A, Tarakeswari S, Thangaraj K, Reddy BM. Role of 14-bp insertion/deletion polymorphism in HLA-G among Indian women with recurrent spontaneous abortions. Tissue Antigens, 2011, 77(2): 131-135.[7] Fabris A, Segat L, Catamo E, Morgutti M, Vendramin A, Crovella S. HLA-G 14 bp deletion/insertion polymorphism in celiac disease. Am J Gastroenterol, 2011, 106(1): 139-144.[8] Jiang Y, Chen S, Jia S, Zhu Z, Gao X, Dong D, Gao Y. Association of HLA-G 3′ UTR 14-bp insertion/deletion polymorphism with hepatocellular carcinoma susceptibility in a Chinese population. DNA Cell Biol, 2011, 30(12): 1027-1032.[9] Veit TD, Vianna P, Scheibel I, Brenol CV, Brenol JC, Xavier RM, Delgado-Canedo A, Gutierrez JE, Brandalize AP, Schuler-Faccini L, Chies JA. Association of the HLA-G 14-bp insertion/deletion polymorphism with juvenile idiopathic arthritis and rheumatoid arthritis. Tissue Antigens, 2008, 71(5): 440-446.[10] Yan WH, Lin A, Chen XJ, Dai MZ, Gan LH, Zhou MY, Zhu M, Shi WW, Liu JM. Association of the maternal 14- bp insertion polymorphism in the HLA-G gene in women with recurrent spontaneous abortions. Tissue Anti-gens, 2006, 68(6): 521-523.[11] Zhu Y, Huo Z, Lai J, Li S, Jiao H, Dang J, Jin C. Case-control study of a HLA-G 14-bp insertion-deletion polymorphism in women with recurrent miscarriages. Scand J Immunol, 2010, 71(1): 52-54.[12] Chen JB, Shi L, Shi L, Yao YF, Yu L, Lin KQ, Tao YF, Yi W, Huang XQ, Sun H, Chu JY. Analysis of the 14 bp insertion/deletion polymorphism in human leukocyte antigen-G gene in Chinese Dai and Han ethnic groups in Yunnan Province. Hereditas, 2010, 32(6): 577-582.[13] Yan WH, Lin A, Li M, Xu HH, Zhang ZP, Wang XX. Analysis of the 14 bp insertion and deletion polymorphism in human leukocyte antigen-G gene in two Chinese ethnic populations. Tissue Antigens, 2008, 71(3): 227-233.[14] Lancaster A, Nelson MP, Meyer D, Thomson G, Single RM. PyPop: a software framework for population genomics: analyzing large-scale multi-locus genotype data. Pac Symp Biocomput, 2003, 8: 514-525.[15] Lancaster AK, Single RM, Solberg OD, Nelson MP, Thomson G. PyPop update-a software pipeline for large-scale multilocus population genomics. Tissue Antigens, 2007, 69(Suppl. 1): 192-197.[16] Yao Y, Shi L, Shi L, Kulski JK, Chen J, Liu S, Yu L, Lin K, Huang X, Tao Y, Tokunaga K, Chu J. The association and differentiation of MHC class I polymorphic Alu insertions and HLA-B/Cw alleles in seven Chinese populations. Tissue Antigens, 2010, 76(3): 194-207.[17] Tao Y, Chen J, Yao Y, Shi L, Lin K, Huang X, Dong Z, Chu J, Shi L. Distribution of HLA-G 14-bp insertion/ deletion polymorphism in six Chinese ethnic groups. Int J Immunogenet, 2012, doi:10.1111/j.1744-313X.2012.01137.x[18] 郭大烈, 董建忠. 中华民族知识通览. 昆明: 云南教育出版社, 2000: 534-535.[19] Yao Y, Shi L, Tao Y, Kulski JK, Lin K, Huang X, Xiang H, Chu J, Shi L. Distinct HLA allele and haplotype distributions in four ethnic groups of China. Tissue Antigens, 2012, 80(5): 452-461.[20] Shi L, Ogata S, Yu JK, Ohashi J, Yu L, Shi L, Sun H, Lin K, Huang XQ, Matsushita M, Horai S, Muramatsu M, Chu JY, Tokunaga K. Distribution of HLA alleles and haplotypes in Jinuo and Wa populations in Southwest China. Hum Immunol, 2008, 69(1): 58-65.[21] Shi L, Shi L, Yao YF, Matsushita M, Yu L, Huang XQ, Yi W, Oka T, Tokunaga K, Chu JY. Genetic link among Hani, Bulang and other Southeast Asian populations: evidence from HLA -A, -B, -C, -DRB1 genes and haplotypes distribution. Int J Immunogenet, 2010, 37(6): 467-475.[22] Shi L, Yao YF, Shi L, Matsushita M, Yu L, Lin QK, Tao YF, Oka T, Chu JY, Tokunaga K. HLA alleles and haplotypes distribution in Dai population in Yunnan province, Southwest China. Tissue Antigens, 2010, 75(2): 159-165.[23] Kulski JK, Martinez P, Longman-Jacobsen N, Wang W, Williamson J, Dawkins RL, Shiina T, Naruse T, Inoko H. The association between HLA-A alleles and an Alu dimorphism near HLA-G. J Mol Evol, 2001, 53(2): 114-123.[24] Kulski JK, Dunn DS. Polymorphic alu insertions within the major histocompatibility complex class I genomic region: a brief review. Cytogenet Genome Res, 2005, 110(1-4): 193-202.[25] Dunn DS, Choy MK, Phipps ME, Kulski JK. The distribution of major histocompatibility complex class I polymorphic Alu insertions and their associations with HLA alleles in a Chinese population from Malaysia. Tissue Antigens, 2007, 70(2): 136-143.[26] Yao Y, Shi L, Shi L, Lin K, Yu L, Sun H, Huang X, Tao Y, Yi W, Liu S, Chu J. The association between HLA-A, -B alleles and major histocompatibility complex class I polymorphic Alu insertions in four populations in China. Tissue Antigens, 2009, 73(6): 575-581.[27] 史磊, 姚宇峰, 史荔, 陶玉芬, 于亮, 黄小琴, 林克勤, 易文, 孙浩, 杨昭庆, 褚嘉祐. 中国壮族和裕固族群体HLA Ⅰ类区域Alu插入多态性及其与HLA-A位点的相关性. 遗传, 2011, 33(2): 138-146.[28] Arnaiz-Villena A, Martinez-Laso J, Serrano-Vela JI, Reguera R, Moscoso J. HLA-G polymorphism and evolution. Tissue Antigens, 2007, 69(Suppl. 1): 156-159. |