[1] Weiss J, Muello K, Victor M, Elsbach P. The role of lipopolysaccharides in the action of the bactericidal/ permeability-increasing neutrophil protein on the bacterial envelope. J Immunol, 1984, 132(6): 3109-3115.[2] Capodici C, Chen S, Sidorczyk Z, Elsbach P, Weiss J. Effect of lipopolysaccharide (LPS) chain length on interactions of bactericidal/permeability-increasing protein and its bioactive 23-kilodalton NH2-terminal fragment with isolated LPS and intact Proteus mirabilis and Escherichia coli. Infect Immun, 1994, 62(1): 259-265.[3] Wilde CG, Seilhamer JJ, McGrogan M, Ashton N, Snable JL, Lane JC, Leong SR, Thornton MB, Miller KL, Scott RW. Bactericidal/permeability-increasing protein and lipopolysaccharide (LPS)-binding protein. LPS binding properties and effects on LPS-mediated cell activation. J Biol Chem, 1994, 269(26): 17411-17416.[4] Iovine NM, Elabach P, Weiss J. An opsonic function of the neutrophil bactericidal/permeability increasing protein depends on both its N- and C-terminal domains. Proc Natl Acad Sci USA, 1997, 94(20): 10973-10978.[5] van der Schaft DW, Toebes EA, Haseman JR, Mayo KH, Griffioen AW. Bactericidal/permeability-increasing protein (BPI) inhibits angiogenesis via induction of apoptosis in vascular endothelial cells. Blood, 2000, 96(1): 176-181.[6] Newman SL, Gootee L, Gabay JE, Selsted ME. Identifica-tion of constituents of human neutrophil azurophil gran-ules that mediate fungistasis against Histoplasma capsulatum. Infect Immun, 2000, 68(10): 5668-5672.[7] Khan AA, Lambert LH Jr, Remington JS, Araujo FG. Recombinant bactericidal/permeability-increasing protein (rBPI21) in combination with sulfadiazine is active against Toxoplasma gondii. Antimicrob Agents Chemother, 1999, 43(4): 758-762.[8] 周红, 郑江, 肖光夏. 猪源杀菌性/通透增加蛋白对革兰氏阴性菌的作用. 中华传染病杂志, 1992, 17(1): 47-48.[9] 周红, 袁建成, 周立新, 郑江, 萧光夏. 杀菌性/通透性增加蛋白中和内毒素作用的体内外研究. 中华医学杂志, 1999, 79(4): 304-305.[10] 周红, 郑江, 秦孝建, 萧光夏. 抗人BPI抗体对猪源BPI体外生物活性的增强作用. 第三军医大学学报, 2002, 24(1): 76-78.[11] 袁树楷. 荣昌猪BPI基因全长cDNA克隆及SNP分析[学位论文]. 重庆: 西南大学, 2007.[12] 曹晓华. 四个猪种间BPI蛋白基因外显子3和4的SNP分析[学位论文]. 重庆: 西南大学, 2008.[13] Tuggle, CK, Thomas JS, Shi XW, Martha AM. Genetic markers for improved disease resistance in animals (BPI). United States Department of Agriculture patents. 2006 no. US 7,070,929 B2, 36 p.[14] Da Silva AS, Valadares GF, Penatti MPA, Brito BG, Da Silva LD. Escherchia coli strains from edema dis-ease: O serogroups, and genes for Shiga toxin, enterotoxins, and F18 fimbriae. Vet Microbiol, 2001, 80(3): 227-233.[15] Meijerink E, Fries R, Vögeli P, Masabanda J, Wigger G, Stricker C, Neuenschwander S, Bertschinger HU, Stranzinger G. Two α (1,2) fucosyltransferase genes on porcine chromosome 6q11 are closely linked to the blood group in-hibitor (S) and Escherichia coli F18 receptor (ECF18R) Loci. Mamm Genome, 1997, 8(10): 736-741.[16] Vögeli P, Meijerink E, Fries R, Neuenschwander S, Vorländer N, Stranzinger G, Bertschinger HU. A molecular test for the detection of E. coli F18 receptors: a Breakthrough in the struggle against edema disease and post-weaning diarrhea in swine. Schweiz Arch Tierheilkd, 1997, 139(11): 479-484.[17] 吴圣龙, 原志伟, 鞠慧萍, 黄雪根, 华金弟, 沈家林, 周冠月, 王建业, 谢恺舟, 陈国宏, 朱国强. 苏太仔猪FUT1基因M307位点多态性与F18大肠杆菌抗病相关性的体外鉴定. 中国预防兽医学报, 2007, 29(10): 783-787.[18] Weiss J, Beckerdite-Quagliata S, Elsbach P. Resistance of gram-negative bacteria to purified bactericidal leukocyte proteins: relation to binding and bacterial lipopolysaccharide structure. J Clin Invest, 1980, 65(3): 619-628.[19] Mannion BA, Weiss J, Elsbach P. Separation of sublethal and lethal effects of polymorphonuclear leukocytes on Escherichia coli. J Clin Invest, 1990, 86(2): 631-641.[20] Takahashi M, Horiuchi Y, Tezuka T. Presence of bacterici |