| [1] Kemphues KJ, Priess JR, Morton DG, Cheng NS. Identification of genes required for cytoplasmic localization in early C. elegans embryos. Cell, 1988, 52(3): 311-320.[2] Namba T, Nakamuta S, Funahashi Y, Kaibuchi K. The role of selective transport in neuronal polarization. Dev Neurobiol, 2011, 71(6): 445-457.[3] Arimura N, Kaibuchi K. Neuronal polarity: from ex-tracellular signals to intracellular mechanisms. Nat Rev Neurosci, 2007, 8(3): 194-205.[4] Watanabe T, Noritake J, Kaibuchi K. Regulation of microtubules in cell migration. Trends Cell Biol, 2005, 15(2): 76-83.[5] Levitan DJ, Boyd L, Mello CC, Kemphues KJ, Stinch-comb DT. Par-2, a gene required for blastomere asymmetry in Caenorhabditis elegans, encodes zinc-finger and ATP-binding motifs. Proc Natl Acad Sci USA, 1994, 91(13): 6108-6112.[6] Etemad-Moghadam B, Guo S, Kemphues KJ. Asymmetri-cally distributed PAR-3 protein contributes to cell polarity and spindle alignment in early C. elegans embryos. Cell, 1995, 83(5): 743-752.[7] Guo S, Kemphues KJ. Par-1, a gene required for estab-lishing polarity in C. elegans embryos, encodes a putative Ser/Thr kinase that is asymmetrically distributed. Cell, 1995, 81(4): 611-620.[8] Hung TJ, Kemphues KJ. PAR-6 is a conserved PDZ do-main-containing protein that colocalizes with PAR-3 in Caenorhabditis elegans embryos. Development, 1999, 126(1): 127-135.[9] Watts JL, Morton DG, Bestman J, Kemphues KJ. The C. elegans par-4 gene encodes a putative serine-threonine kinase required for establishing embryonic asymmetry. Development, 2000, 127(7): 1467-1475.[10] Morton DG, Shakes DC, Nugent S, Dichoso D, Wang WF, Golden A, Kemphues KJ. The Caenorhabditis elegans par-5 gene encodes a 14-3-3 protein required for cellular asymmetry in the early embryo. Dev Biol, 2002, 241(1): 47-58.[11] Böhm H, Brinkmann V, Drab M, Henske A, Kurzchalia TV. Mammalian homologues of C. elegans PAR-1 are asym-metrically localized in epithelial cells and may influence their polarity. Curr Biol, 1997, 7(8): 603-606.[12] Zhou Y. Cortical development and asymmetric cell divi-sions. Front Biol, 2012, 7(4): 297-306.[13] Lin D, Edwards AS, Fawcett JP, Mbamalu G, Scott JD, Pawson T. A mammalian PAR-3-PAR-6 complex implicated in Cdc42/Rac1 and aPKC signalling and cell polarity. Nat Cell Biol, 2000, 2(8): 540-547.[14] Watts JL, Etemad-Moghadam B, Guo S, Boyd L, Draper BW, Mello CC, Priess JR, Kemphues KJ. Par-6, a gene involved in the establishment of asymmetry in early C. elegans embryos, mediates the asymmetric localization of PAR-3. Development, 1996, 122(10): 3133-3140.[15] Suzuki A, Akimoto K, Ohno S. Protein kinase C λ/ι (PKC λ/ι): a PKC isotype essential for the development of mul-ticellular organisms. J Biochem, 2003, 133(1): 9-16.[16] Komada M, Soriano P. Hrs, a FYVE finger protein local-ized to early endosomes, is implicated in vesicular traffic and required for ventral folding morphogenesis. Genes Dev, 1999, 13(11): 1475-1485.[17] Farkas LM, Huttner WB. The cell biology of neural stem and progenitor cells and its significance for their prolif-eration versus differentiation during mammalian brain development. Curr Opin Cell Biol, 2008, 20(6): 707-715.[18] Nadarajah B, Alifragis P, Wong ROL, Parnavelas JG. Neuronal migration in the developing cerebral cortex: ob-servations based on real-time imaging. Cereb Cor-tex, 2003, 13(6): 607-611.[19] Smart IHM. Three dimensional growth of the mouse iso-cortex. J Anat, 1983, 137(4): 683-694.[20] Noctor SC, Flint AC, Weissman TA, Dammerman RS, Kriegstein AR. Neurons derived from radial glial cells es-tablish radial units in neocortex. Nature, 2001, 409(6821): 714-720.[21] Noctor SC, Martínez-Cerdeño V, Kriegste |
